Physician

Neonat/Perinatology

Dr. Erik N. Meyers, MD

Dr. Erik N. Meyers works in the field of Neonat/Perinatology. He researched medicine at University of California San Diego School of Medicine. University of California San Diego School of Medicine is ranked 17 in research and 19 in primary care medicine Erik Meyers has written publications. He has 20 publications published with PubMed. The lastest publication is: 'Analysis of Fgf8 gene function in vertebrate development.'

Publications

The bone morphogenetic protein antagonist noggin regulates mammalian cardiac morphogenesis.
Loss of Gbx2 results in neural crest cell patterning and pharyngeal arch artery defects in the mouse embryo.
Chorioallantoic fusion defects and embryonic lethality resulting from disruption of Zfp36L1, a gene encoding a CCCH tandem zinc finger protein of the Tristetraprolin f...
Cre-mediated excision of Fgf8 in the Tbx1 expression domain reveals a critical role for Fgf8 in cardiovascular development in the mouse.
Tbx1 is regulated by tissue-specific forkhead proteins through a common Sonic hedgehog-responsive enhancer.
Conditional inactivation of Fgf4 reveals complexity of signalling during limb bud development.
Analysis of Fgf8 gene function in vertebrate development.
An Fgf8 mutant allelic series generated by Cre- and Flp-mediated recombination.
Differences in left-right axis pathways in mouse and chick: functions of FGF8 and SHH.
Targeted disruption of Fgf8 causes failure of cell migration in the gastrulating mouse embryo.
An FGF autocrine loop initiated in second heart field mesoderm regulates morphogenesis at the arterial pole of the heart.
FGF8 is required for cell survival at distinct stages of nephrogenesis and for regulation of gene expression in nascent nephrons.
Sonic hedgehog is required for cardiac outflow tract and neural crest cell development.
Fgf8 is required for anterior heart field development.
Fgf8 is required for pharyngeal arch and cardiovascular development in the mouse.
A genetic link between Tbx1 and fibroblast growth factor signaling.
BMP receptor IA is required in mammalian neural crest cells for development of the cardiac outflow tract and ventricular myocardium.
Intracardiac septation requires hedgehog-dependent cellular contributions from outside the heart.
Independent requirements for Hedgehog signaling by both the anterior heart field and neural crest cells for outflow tract development.

Schools

University Of California San Diego School Of Medicine

Doctors Specialties

Neonatal Medicine
Neonat/Perinatology

Accepted Insurances

Education

UCSF Medical Center
University of California San Diego School of Medicine

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