Associate Professor, Medicine, Perelman School of Medicine
Gastroenterology
Dr. Ben Z. Stanger, MD
|
|
Give Review
Doctor Ben Z. Stanger, MD practices Gastroenterology. He graduated from Harvard Medical School. Harvard Medical School is a one of the best medicals schools. Harvard Medical School is ranked 1 in research and 12 in primary care medicine He has 2 awards "CMS Meaningful Use Stage 1 Certification" and "Elected Member". Doctor Ben Z. Stanger, MD is a published doctor as well. He has 60 publications published. The latest was: The death domain kinase RIP mediates the TNF-induced NF-kappaB signal. Doctor Ben Z. Stanger, MD accepts Medicare payments and is registered with Medicare.gov.
Publications
- Echoes of the embryo: using the developmental biology toolkit to study cancer.
- Reprogrammed Stomach Tissue as a Renewable Source of Functional β Cells for Blood Glucose Regulation.
- Probing hepatocyte heterogeneity.
- The Poly(C) binding protein Pcbp2, and its retrotransposed derivative Pcbp1, are independently essential to mouse development.
- Induction of T-cell Immunity Overcomes Complete Resistance to PD-1 and CTLA-4 Blockade and Improves Survival in Pancreatic Carcinoma.
- Adult hepatocytes are generated by self-duplication rather than stem cell differentiation.
- Cell competition in vertebrate organ size regulation.
- Akt-Dependent Metabolic Reprogramming Regulates Tumor Cell Histone Acetylation.
- Cytokinesis defines a spatial landmark for hepatocyte polarization and apical lumen formation.
- Stromal elements act to restrain, rather than support, pancreatic ductal adenocarcinoma.
- Hippo pathway activity influences liver cell fate.
- De Novo Formation of Insulin-Producing "Neo-β Cell Islets" from Intestinal Crypts.
- Liver cell reprogramming: Parallels with iPSC biology.
- Pdx1 Maintains β Cell Identity and Function by Repressing an α Cell Program.
- Detection of Circulating Pancreas Epithelial Cells in Patients with Pancreatic Cystic Lesions.
- Antiviral Autophagy Restricts Rift Valley Fever Virus Infection and Is Conserved from Flies to Mammals.
- Interleukin-6 is required for pancreatic cancer progression by promoting MAPK signaling activation and oxidative stress resistance.
- LIN28B promotes growth and tumorigenesis of the Intestinal epithelium via Let-7.
- Platelets and tumor cells: a new form of border control.
- Control of cell identity in pancreas development and regeneration.
- Hippo Signaling Regulates Differentiation and Maintenance in the Exocrine Pancreas.
- Advances in acute and chronic pancreatitis: from development to inflammation and repair.
- Quit your YAPing: a new target for cancer therapy.
- Tumor-derived granulocyte-macrophage colony-stimulating factor regulates myeloid inflammation and T cell immunity in pancreatic cancer.
- Ngn3(+) endocrine progenitor cells control the fate and morphogenesis of pancreatic ductal epithelium.
- Lineage tracing demonstrates no evidence of cholangiocyte epithelial-to-mesenchymal transition in murine models of hepatic fibrosis.
- Facultative stem cells in liver and pancreas: fact and fancy.
- Notch1 functions as a tumor suppressor in a model of K-ras-induced pancreatic ductal adenocarcinoma.
- Intrahepatic bile ducts develop according to a new mode of tubulogenesis regulated by the transcription factor SOX9.
- Inhibition of gamma-secretase activity inhibits tumor progression in a mouse model of pancreatic ductal adenocarcinoma.
- HNF4A and diabetes: injury before insult?
- Organ size determination and the limits of regulation.
- Regeneration in liver and pancreas: time to cut the umbilical cord?
- The fringe molecules induce endocrine differentiation in embryonic endoderm by activating cMyt1/cMyt3.
- Genetics and biology of pancreatic ductal adenocarcinoma.
- Dissecting the cellular origins of pancreatic cancer.
- Pten constrains centroacinar cell expansion and malignant transformation in the pancreas.
- Direct regulation of Intestinal fate by Notch.
- Notch signaling controls multiple steps of pancreatic differentiation.
- YAP regulates S-phase entry in endothelial cells.
- Doublecortin-like kinase 1 is elevated serologically in pancreatic ductal adenocarcinoma and widely expressed on circulating tumor cells.
- Function of GATA Factors in the Adult Mouse Liver.
- The p130 Isoform of Angiomotin Is Required for Yap-Mediated Hepatic Epithelial Cell Proliferation and Tumorigenesis.
- Notch signaling is required for the generation of hair cells and supporting cells in the mammalian inner ear.
- Protection against Fas-dependent Th1-mediated apoptosis by antigen receptor engagement in B cells.
- The biology of organ size determination.
- Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.
- The death domain kinase RIP mediates the TNF-induced NF-kappaB signal.
- Looking beneath the surface: the cell death pathway of Fas/APO-1 (CD95).
- RIP and FADD: two "death domain"-containing proteins can induce apoptosis by convergent, but dissociable, pathways.
- Dissociations among structural-perceptual, lexical-semantic, and event-fact memory Systems in Alzheimer, amnesic, and normal subjects.
- The molecular mechanism of FasL-mediated cytotoxicity by CD4+ Th1 clones.
- RIP: a novel protein containing a death domain that interacts with Fas/APO-1 (CD95) in yeast and causes cell death.
- Fas ligand-mediated cytotoxicity is directly responsible for apoptosis of normal CD4+ T cells responding to a bacterial superantigen.
- Organ size is limited by the number of embryonic progenitor cells in the pancreas but not the liver.
- Molecular mechanisms of bile duct development.
- Molecular biology of pancreatic ductal adenocarcinoma progression: aberrant activation of developmental pathways.
- Notch signaling controls liver development by regulating biliary differentiation.
- Notch signaling promotes airway mucous metaplasia and inhibits alveolar development.
Schools
Harvard Medical School
UCSF
Massachusetts General Hospital
Procedures Preformed
- Colonoscopy, Proctosigmoidoscopy, and Sigmoidoscopy
- Endoscopy (Esophagus, Stomach, Small Intestine)
Conditions Treated
- Abdominal Pain
- Anemia
- Barrett's Esophagus
- Benign Neoplasm of the Digestive System
- View All
Doctors Specialties
- Gastroenterology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Elected Member
Education
-
Massachusetts General Hospital -
Harvard Medical School
Hospital
-
Hospital of the University of Pennsylvania -
Penn Presbyterian Medical Center
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1730179813 |
| Last Name Of The Provider | STANGER |
| First Name Of The Provider | BEN |
| View All |
Similar Doctors
|
|
Dr. James D. Lewis, MD
 This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Oleh Haluszka, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Nnaemeka U. Anyadike, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Rebecca A. Matro, MD |
|
|
Dr. Bruce P. Gelman, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Gary D. Wu, MD
This Doctor is OpenDoctor Recommended.
|
|
Dr. Henry Parkman, MD
This Doctor is OpenDoctor Recommended.
 This doctor has more publications
|
|
Dr. Michael C. Dimarino, MD
This Doctor is OpenDoctor Recommended.
|